Classifying the type of pain you have in the pain clinic is an essential first step to make, allowing you to choose the correct pain killer/reliever, and achieving better pain management.

Nociceptive Pain arises from the stimulation of specific pain receptors. These receptors can respond to heat, cold, vibration, stretch and chemical stimuli released from damaged cells.

Non Nociceptive Pain arises from within the peripheral and central nervous system. Specific receptors do not exist here, with pain being generated by nerve cell dysfunction.

Somatic Pain

Source: tissues such as skin, muscle, joints, bones, and ligaments – often known as musculo-skeletal pain.

Receptors activated: specific receptors (nociceptors) for heat, cold, vibration, stretch(muscles), inflammation (e.g. cuts and sprains which cause tissue disruption), and oxygen starvation (ischaemic muscle cramps).

Characteristics: often sharp and well localized, and can often be reproduced by touching or moving the area or tissue involved.

Visceral Pain

Source: internal organs of the main body cavities. There are three main cavities- thorax (heart and lungs), abdomen (liver, kidneys, spleen and bowels),pelvis (bladder, womb, and ovaries).

Receptors activated: specific receptors (nociceptors) for stretch, inflammation, and oxygen starvation (ischemia).

Characteristics: often poorly localized, and may feel like a vague deep ache, sometimes being cramping or colicky in nature. It frequently produces referred pain to the back, with pelvic pain referring pain to the lower back, abdominal pain referring pain to the mid-back, and thoracic pain referring pain to the upper back.

Nerve Pain

Source: from within the nervous system itself – also known as pinched nerve, trapped nerve. The pain may originate from the peripheral nervous system (the nerves between the tissues and the spinal cord), or from the central nervous system (the nerves between the spinal cord and the brain)

Causes- may be due to any one of the following processes:

Nerve Degeneration: multiple sclerosis, stroke, brain hemorrhage or oxygen starvation

Nerve Pressure: trapped nerve

Nerve Inflammation – torn or slipped disc

Nerve Infection – shingles and other viral infections

Receptors activated: the nervous system does not have specific receptors for pain (nonnocicpetive). Instead, when a nerve becomes injured by one of the processes named above, it becomes electrically unstable, firing off signals in a completely inappropriate, random, and disordered fashion.

Characteristics: These signals are then interpreted by the brain as pain, and can be associated with signs of nerve malfunction such as hypersensitivity (touch, vibration, hot and cold), tingling, numbness, and weakness. There is often referred pain to an area where that nerve would normally supply e.g. sciatica from a slipped disc irritating the L5 spinal nerve produces pain down the leg to the outside shin and big toe i.e. the normal territory in the leg supplied by the L5 spinal nerve. Spinal nerve root pain is also often associated with intense itching in the distribution of a particular dermatome. People often describe nerve pain is often described as lancinating, shooting, burning, and hypersensitive.

Sympathetic Pain

Source: due to possible over-activity sympathetic nervous system, and central/peripheral nervous system mechanisms. The sympathetic nervous system controls blood flow to tissues such as skin and muscle, sweating by the skin, and the speed and responsiveness of the peripheral nervous system.

Causes: occurs more commonly after fractures and soft tissue injuries of the arms and legs, and these injuries may lead to Complex Regional Pain Syndrome (CRPS).

Receptors activated: like nerve pain there are no specific pain receptors (non nociceptive).

Characteristics: presents as extreme hypersensitivity in the skin around the injury and also peripherally in the limb, and is associated with abnormalities of sweating and temperature control in the area. The limb is usually so painful, that the sufferer refuses to use it, causing secondary problems after a period of time with muscle wasting, joint contractures, and osteoporosis of the bones. It is possible that the syndrome is initiated by trauma to small peripheral nerves close to the injury.



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